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Journal of Neuroimmunology
Volume 178, Issue 1
, Pages 1-8
, September 2006
IL-10 is essential for disease protection following intranasal peptide administration in the C57BL/6 model of EAE
References
- . Role of interleukin 10 in specific immunotherapy. J. Clin. Invest. 1998;102:98–106
- . A range of CD4 T cell tolerance: partial inactivation to organ-specific antigen allows nondestructive thyroiditis or insulitis. Immunity. 1997;7:255–271
- . Hierarchy in the ability of T cell epitopes to induce peripheral tolerance to antigens from myelin. Eur. J. Immunol. 1998;28:1251–1261
- . Induction of chronic relapsing experimental allergic encephalomyelitis in Biozzi mice. J. Neuroimmunol. 1990;28:261–267
- . IL-10 is critical in the regulation of autoimmune encephalomyelitis as demonstrated by studies of IL-10- and IL-4-deficient and transgenic mice. J. Immunol. 1998;161:3299–3306
- . Peptide-induced T cell regulation of experimental autoimmune encephalomyelitis: a role for interleukin-10. Int. Immunol. 1999;11:1625–1634
- . T regulatory cells 1 inhibit a Th2-specific response in vivo. J. Immunol. 2000;165:4848–4853
- . Protection of nonobese diabetic mice from diabetes by intranasal or subcutaneous administration of insulin peptide B-(9–23). Proc. Natl. Acad. Sci. U. S. A. 1996;93:956–960
- . Glatiramer acetate (Copaxone) induces degenerate, Th2-polarized immune responses in patients with multiple sclerosis. J. Clin. Invest. 2000;105:967–976
- . Inflammation in EAE: role of chemokine/cytokine expression by resident and infiltrating cells. Neurochem. Res. 1996;21:511–525
- . Disease modifying therapies in multiple sclerosis: report of the Therapeutics and Technology Assessment Subcommittee of the American Academy of Neurology and the MS Council for Clinical Practice Guidelines. Neurology. 2002;58:169–178
- . Interleukin-10 induces a long-term antigen-specific anergic state in human CD4+ T cells. J. Exp. Med. 1996;184:19–29
- . Leukocytes infiltrating the pancreatic islets of nonobese diabetic mice are transformed into inactive exiles by combinational anti-cell adhesion therapy. J. Leukoc. Biol. 2001;70:510–517
- . Peptide-based therapeutic vaccines for allergic and autoimmune diseases. Nat. Med. 2005;11:S69–S76
- . IFN-alpha and IL-10 induce the differentiation of human type 1 T regulatory cells. J. Immunol. 2001;166:5530–5539
- . Regulatory Th2-type T cell lines against insulin and GAD peptides derived from orally- and nasally-treated NOD mice suppress diabetes. J. Autoimmun. 1999;12:251–258
- . Treatment of experimental autoimmune encephalomyelitis with genetically modified memory T cells. J. Exp. Med. 1997;186:159–164
- . Inhibition of experimental autoimmune encephalomyelitis by inhalation but not oral administration of the encephalitogenic peptide: influence of MHC binding affinity. Int. Immunol. 1993;5:1159–1165
- . Effect of T-cell peptides derived from Fel d 1 on allergic reactions and cytokine production in patients sensitive to cats: a randomised controlled trial. Lancet. 2002;360:47–53
- . Epitope-specific immunotherapy induces immune deviation of proinflammatory T cells in rheumatoid arthritis. Proc. Natl. Acad. Sci. U. S. A. 2004;101:4228–4233
- . Superantigen modulation of experimental allergic encephalomyelitis: activation of energy determines outcome. J. Immunol. 1994;152:2051–2059
- . Peptides containing a dominant T-cell epitope from red cell band 3 have in vivo immunomodulatory properties in NZB mice with autoimmune hemolytic anemia. Blood. 2003;102:3800–3806
- . Role for IL-10 in suppression mediated by peptide-induced regulatory T cells in vivo. J. Immunol. 2003;170:1240–1248
- . Nasal administration of glutamate decarboxylase (GAD 65) peptides induces Th2 responses and prevents murine insulin-dependent diabetes. J. Exp. Med. 1996;183:1561–1567
- . Role of interleukin-10 in the induction and function of natural and antigen-induced regulatory T cells. J. Autoimmun. 2003;20:273–275
- . Vaccines for the treatment of autoimmune diseases. In: Levine Myron M, Rino Rappuoli JBK, Liu Margaret, Good Michael editor. New Generation Vaccines. New York: Marcel Dekker Inc.; 2004;p. 1047–1056
- . Suppression of ongoing experimental allergic encephalomyelitis (EAE) in Lewis rats: synergistic effects of myelin basic protein (MBP) peptide 68–86 and IL-4. Clin. Exp. Immunol. 2000;120:526–531
- . A comparison of the mechanisms of action of interferon beta and glatiramer acetate in the treatment of multiple sclerosis. Clin. Ther. 2002;24:1998–2021
PII: S0165-5728(06)00210-4
doi: 10.1016/j.jneuroim.2006.05.030
© 2006 Elsevier B.V. All rights reserved.
« Previous
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Journal of Neuroimmunology
Volume 178, Issue 1
, Pages 1-8
, September 2006
